简介:Theground-stateandthermodynamicpropertiesofquantummixed-spinchainsof1/2-1/2-1-1and3/2-3/2-1-1areinvestigatedbyaquantumMonteCarlosimulationwiththeloop-clusteralgorithm.For1/2-1/2-1-1chain,wefindithastwophasesseparatedbyanenergy-gapvanishingpointintheground-state.For3/2-3/2-1-1chain,thenumericalresultsshowtwoenergy-gapvanishingpointsisolatedbydifferentphasesinitsground-state.Ourcalculationsindicatethatallthesegroundstatephasescanbeunderstoodbymeansofvalence-bond-solidpicture,andthethermodynamicbehavioratfinitetemperaturesiscontinuousasafunctionofparameterα=J2/J1.
简介:ThesplittingofpotentialenergylevelsforgroundstateX2ΠgofOx2(x=+1,1)underspin–orbitcoupling(SOC)hasbeencalculatedbyusingthespin–orbit(SO)multi-configurationquasi-degenerateperturbationtheory(SO-MCQDPT).TheirMurrell–Sorbie(M–S)potentialfunctionsaregained,andthenthespectroscopicconstantsforelectronicstates2Π1/2and2Π3/2arederivedfromtheM–Sfunction.TheverticalexcitationenergiesforOx2(x=+1,1)areν[O+12(2Π3/2→X2Π1/2)]=195.652cm1,andν[O12(2Π1/2→X2Π3/2)]=182.568cm1,respectively.Allthespectroscopicdataforelectronicstates2Π1/2and2Π3/2aregivenforthefirsttime.
简介:采用球衣菌(Sphaerotilwsnatans)FQ32为生物吸附剂,研究了Pb^2+初始浓度、吸附剂用量、菌龄、pH值、温度和吸附时间等理化因素对球衣菌吸附硝酸铅溶液模拟废水中Pbc的影响。结果表明,球衣菌在Pb^2+初始质量浓度为50mg/L、吸附剂0.6g/L用量、菌龄32h、pH=7、温度30℃、吸附时间60min的条件下,对Pb^2+的吸附率为89.98%,吸附量为74.98mg/g;该吸附过程是一个快速的过程,在吸附5min时,吸附量已达总吸附量的94%,90min达到吸附平衡。球衣菌对Pb^2+的吸附基本为细胞表面的被动吸附,吸附条件对吸附效率有较大影响。
简介:ConsideringthatKBe2BO3F2(KBBF)isanoutstandingdeepultravioletcrystalwhichcangeneratetheshortestwavelengthsinsecondharmonicgenerationandsum-frequencygeneration,wereportthedeterminationofthenonlinearopticalcoefficientsoftheKBBFcrystal.Thed11coefficientwasdeterminedtobe0.49pm/VbytheMakerfringesmethodatthewavelength1064nm,whichisinagreementwellwiththetheoreticalvalue.
简介:摘要LINC01018和CDK6均与恶性肿瘤的发生发展相关。恶性肿瘤中LINC01018表达下调、CDK6上调,且与恶性程度有关;生物信息学提示CDK6启动子存在E2F1结合位点,而LINC01018与E2F1可能有相互作用。由此推测,恶性肿瘤中LINC01018表达降低,LINC01018与E2F1相互作用后活化E2F1,促进CDK6转录激活,影响肿瘤增殖、侵袭和转移,从而揭示恶性肿瘤发生发展及调控的分子机制,为其治疗提供新思路和证据。
简介:摘要LINC01018和CDK6均与恶性肿瘤的发生发展相关。恶性肿瘤中LINC01018表达下调、CDK6上调,且与恶性程度有关;生物信息学提示CDK6启动子存在E2F1结合位点,而LINC01018与E2F1可能有相互作用。由此推测,恶性肿瘤中LINC01018表达降低,LINC01018与E2F1相互作用后活化E2F1,促进CDK6转录激活,影响肿瘤增殖、侵袭和转移,从而揭示恶性肿瘤发生发展及调控的分子机制,为其治疗提供新思路和证据。